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Human associative learning Lecture 2 Intro to human associative learning pt 2 Prof Mike Le Pelley [email protected] PSYC 2081 The Hats of Power • Post thoughts, insights, jokes, memes,

observations etc to the Moodle forum • Best entries will win one of four Hats of Power Summary of Lecture 1 • Contingency: How strongly are events related? • Learning is influenced by contingency (as you would hope!) • But that’s not the whole story – for example – Learning is gradual even though ΔP is constant; “learning curves” – When we learn about multiple cues at the same time, we don’t learn

about each one individually based on its contingency – Instead cues compete with each other  Blocking as an example of “cue competition” • Learning is not the same as calculating contingency. So what

are people doing when they learn? • We can understand learning as the gradual formation of an

association (a link) between representations An alternative view: Association formation CS cue US outcome • What determines whether (and how fast) this association

strengthens? • Proposed by Rescorla & Wagner (1972) • Surprise is the key to learning – We only learn when something happens that we don’t expect  We learn when we are surprised by something – If everything occurs as expected, we don’t learn anything new  We don’t learn if we are not surprised • Rescorla & Wagner captured this idea in an equation, providing a

simple but powerful model of the learning process The Rescorla-Wagner model Robert

Rescorla Allan

Wagner ∆V

=

α β

( λ

– ΣV ) Salience

of cue Salience

of

outcome Observed

magnitude

of outcome Expected

magnitude

of outcome Σ means “sum” So ΣV means “summed

associative strength of all cues” ∆ means “change” So ∆V means “change in

associative strength” i.e., learning cue outcome V V = predictive Value (or associative strength) Amount of

learning The Rescorla-Wagner model ∆V

=

α β

( λ

– ΣV ) Prediction error: “Surprisingness” of

outcome • When prediction error is zero, there is no learning • Learning

=

Reduction in PE • Learning

=

Things becoming more expected, and less surprising Salience

of cue Salience

of

outcome Observed

magnitude

of outcome Expected

magnitude

of outcome Amount of

learning The Rescorla-Wagner model cue outcome V V = predictive Value (or associative strength) V = 0.4 ∆V

=

α

β

( λ

– ΣV ) Associative

strength Salience

of cue Salience of

outcome Observed

magnitude of

outcome Expected

magnitude of

outcome ∆Vvege =

0.5

x

0.8

( 1 – 0 )

=

0.4 Vvege =

0 Vvege =

0.4 0.5 0.8 1Let’s assume Vege illness Large

prediction

error V = 0.64 ∆V

=

α

β

( λ

– ΣV ) ∆Vvege =

0.5

x

0.8

( 1 – 0.4 )

=

0.24 Vvege =

0.4 Vvege =

0.64 Vege illness Smaller

prediction

error Associative

strength Salience

of cue Salience of

outcome Observed

magnitude of

outcome Expected

magnitude of

outcome 0.5 0.8 1Let’s assume Trial 0:

Vvege =

0 Trial 1:

Vvege =

0.40 Trial 2:

Vvege =

0.64 Trial 3:

Vvege =

0.78 Trial 4:

Vvege =

0.87 Trial 5:

Vvege =

0.92 0 0.5 1 0 5 10 15 As so cia tiv e

st re ng th Trial ∆V

=

α

β

( λ

– ΣV ) SURPRISING NOT SURPRISING Associative

strength Salience

of cue Salience of

outcome Observed

magnitude of

outcome Expected

magnitude of

outcome 0.5 0.8 1Let’s assume V = 0.99 Vege illness 0 0.5 1 0 5 10 15 As so cia tiv e

st re ng th Trial ∆V

=

α

β

( λ

– ΣV ) Associative

strength Salience

of cue Salience of

outcome Observed

magnitude of

outcome Expected

magnitude of

outcome ∆Vvege =

0.5

x

0.8

( 0 – 0.99 )

=

–0.40 Vvege =

0.99 Vvege =

0.59 0.5 0.8 0Let’s assume Negative prediction

error V = 0.59 Vege illness V = 0.35 ∆V

=

α

β

( λ

– ΣV ) ∆Vvege =

0.5

x

0.8

( 0 – 0.59 )

=

–0.24 Vvege =

0.59 Vvege =

0.35 Vege illness Smaller negative prediction

error Associative

strength Salience

of cue Salience of

outcome Observed

magnitude of

outcome Expected

magnitude of

outcome 0.5 0.8 0Let’s assume 00.5 1 0 5 10 15 20 25 As so cia tiv e

st re ng th Trial Acquisition Extinction Acquisition and extinction SURPRISING NOT SURPRISING NOT SURPRISINGSURPRISING ∆V

=

α

β

( λ

– ΣV ) Salience

of cue Salience of

outcome Observed

magnitude

of outcome Expected

magnitude

of outcome 0 0.5 1 0 5 10 15 As so cia tiv e

st re ng th Trial Associative

strength α = 0.5, β = 0.8 α = 0.5, β = 0.2 α = 0.2, β = 0.8 -80 -60 -40 -20 0 20 40 60 80 0 5 10 15 20 25 30 35 40 Ju dg m en t r at in g Trial Lopez & Shanks (unpublished) See Shanks 1995, ch2, p32 Rescorla-Wagner and contingency People • Learning is gradual, even though contingency (ΔP) does not change – “Learning curves” • The Rescorla-Wagner model also shows this pattern ∆P = 0.5 ∆P = -0.5 -0.6 -0.4 -0.2 0 0.2 0.4 0.6 0 5 10 15 20 25 30 35 40 As so cia tiv e

st re ng th Trial RW ∆P = 0.5 ∆P = -0.5 01 2 3 4 Bread Dates Ju dg m en t r at in g Aitken, Larkin & Dickinson (2000, Expt 1) Phase 1 Phase 2 Test Apple → allergy Apple & Bread → allergy Bread? Carrot & Dates → allergy Dates? Rescorla-Wagner and blocking • Previous learning about apples blocks subsequent learning about bread • Cues are not learned about

independently • Instead, cues compete with each other 00.5 1 0 5 10 15 20 As so cia tiv e

st re ng th Trial ∆Vbread :

(λ – ΣV) Vapple Rescorla-Wagner and blocking Phase 1 Phase 2 Test Apple → allergy Apple & Bread → allergy Bread? Carrot & Dates → allergy Dates? 00.5 1 0 5 10 15 20 As so cia tiv e

st re ng th Trial ∆Vbread :

(1 – ΣV) Vapple Rescorla-Wagner and blocking Phase 1 Phase 2 Test Apple → allergy Apple & Bread → allergy Bread? Carrot & Dates → allergy Dates? 00.5 1 0 5 10 15 20 As so cia tiv e

st re ng th Trial ∆Vbread :

(1 – [Vapple + Vbread]) Vapple Rescorla-Wagner and blocking Phase 1 Phase 2 Test Apple → allergy Apple & Bread → allergy Bread? Carrot & Dates → allergy Dates? 00.5 1 0 5 10 15 20 As so cia tiv e

st re ng th Trial Vapple Rescorla-Wagner and blocking Outcome not

surprising ∆Vbread :

(1 – [0.85 + Vbread]) Phase 1 Phase 2 Test Apple → allergy Apple & Bread → allergy Bread? Carrot & Dates → allergy Dates? 00.5 1 0 5 10 15 20 As so cia tiv e

st re ng th Trial Vbread Vapple Outcome not

surprising Rescorla-Wagner and blocking = 0.15 Small

prediction

error ∆Vbread :

(1 – [0.85 + 0]) Phase 1 Phase 2 Test Apple → allergy Apple & Bread → allergy Bread? Carrot & Dates → allergy Dates? Allergy Apples Bread Phase 1 Phase 2 Test Apple → allergy Apple & Bread → allergy Bread? Carrot & Dates → allergy Dates? Rescorla-Wagner and blocking Allergy Carrot Dates Phase 1 Phase 2 Test Apple → allergy Apple & Bread → allergy Bread? Carrot & Dates → allergy Dates? Rescorla-Wagner and blocking 01 2 3 4 Bread Dates Ju dg m en t r at in g Aitken, Larkin & Dickinson (2000, Expt 1) Phase 1 Phase 2 Test Apple → allergy Apple & Bread → allergy Bread? Carrot & Dates → allergy Dates? • People learn a

weaker

association for

bread than dates • And so does the

RW model! 0 0.2 0.4 0.6 0.8 Bread Dates As so cia tiv e

st re ng th People RW Rescorla-Wagner and blocking Summary so far • Lecture 1:

Learning

=

Calculating contingency? • A different view:

Learning

=

Forming association between

representations • The Rescorla-Wagner model – Surprise drives association formation (learning) – RW is one model of association formation – there are many others  RW is probably the most influential model of associative learning  A simple, intuitive account – but very powerful • E.g., explains effects of contingency, learning curves, and cue

competition (blocking)  Still often used as a “default” view of learning An alternative view “Many theorists maintain a belief in a learning mechanism in

which links between mental representations are formed

automatically… We conclude that learning is the consequence

of propositional reasoning processes that cooperate with the

unconscious processes involved in memory retrieval” An alternative view The link-based approach (e.g., Rescorla-Wagner) ILLV The propositional approach Last time I ate Vegemite I was ill. Perhaps Vegemite makes me ill. The propositional approach Last 2 times I ate Vegemite I was ill. Fairly sure Vegemite makes me ill. 0 0.5 1 0 5 10 15 St re ng th

o f b el ie f Trial The propositional approach • The “thinking about it” approach • Remember what went with what in the past, try and

work out most likely explanation

Blocking 0 1 2 3 4 Bread Dates Ju dg m en t r at in g • Previous learning about effectiveness of apples

blocks subsequent learning

about bread • Cues compete with each

other Phase 1 Phase 2 Test Apple → allergy Apple & Bread → allergy Bread? Carrot & Dates → allergy Dates? Aitken, Larkin & Dickinson (2000, Expt 1) Propositions and blocking • Remember what went with what in the past, try and

work out most likely explanation

“If apple and bread together cause the outcome to occur with

the same probability as apple alone, this implies that bread is

not a cause of the outcome.” Phase 1 Phase 2 Test Apple → allergy Apple & Bread → allergy Bread? Carrot & Dates → allergy Dates? Blocking and trial order • According to the propositional account, order of the trials doesn’t matter • Should get blocking in the standard (forward) procedure, and in a backward

procedure Phase 1 Phase 2 Test A → outcome A + B → outcome B? C + D → outcome D? “If A and B together cause the outcome to occur with the same probability and

magnitude as A alone, this implies that B is not a cause of the outcome.” FO RW AR D Proposition Blocking BA CK W AR D Phase 1 Phase 2 Test A + B → outcome A → outcome B? C + D → outcome D? Blocking • In the Forward case, RW model says that blocking occurs because the outcome

is less surprising on “A+B” trials than “C+D” trials • In the backward case, the outcome is equally surprising on “A+B” trials and

“C+D” trials • So RW model predicts no blocking in backward condition Blocking and trial order Phase 1 Phase 2 Test A → outcome A + B → outcome B? C + D → outcome D?FO RW AR D Proposition Blocking BA CK W AR D Phase 1 Phase 2 Test A + B → outcome A → outcome B? C + D → outcome D? Blocking RW Blocking No blocking B D 0 2 4 6 8 Ju dg m en t r at in g ✱• We’ve already seen

blocking in the

“Forward” case – e.g., Aitken, Larkin &

Dickinson (2000), see

Lecture 1 • And it also occurs in the

“Backward” case! – e.g., Wasserman & Berglan (1998) • Consistent with propositional

account, but not with RW Blocking and trial order Phase 1 Phase 2 Test A → outcome A + B → outcome B? C + D → outcome D?FO RW AR D Proposition BA CK W AR D Phase 1 Phase 2 Test A + B → outcome A → outcome B? C + D → outcome D? Blocking Blocking RW Blocking No blocking “If A and B together cause the outcome to occur with the same

probability and magnitude as A alone, this implies that B is not

a cause of the outcome.” • In order to draw this inference, you need to know that B has

been paired with the outcome – Inferences based on memories of A→outcome and AB→outcome – B is paired with the outcome, but does not cause the outcome Blocking of memory Phase 1 Phase 2 Test A → outcome AB → outcome B? Causality Memory Propositional account Blocking of causal

judgement Good memory of

outcome Link-based account

(e.g. RW) Blocking of causal

judgement Blocking of memory

of outcome Phase 1 Phase 2 Test A → outcome AB → outcome B? Blocking of memory Phase 1 Phase 2 Test A → outcome AB → outcome B? A B outcome • RW:

Blocked cue does not form a

strong link with the outcome – People don’t encode that B was ever

paired with the outcome Blocking of memory Phase 1 Phase 2 Test A → outcome AB → outcome B? Blocking of memory Causality Memory Propositional account Blocking of causal

judgement Good memory of

outcome Link-based account

(e.g. RW) Blocking of causal

judgement Blocking of memory

of outcome Mitchell, Lovibond, Minard & Lavis (2006) Mr X ate Apple He suffered Xianethis Mitchell, Lovibond, Minard & Lavis (2006) Mr X ate Peach He suffered Daryosis Mitchell, Lovibond, Minard & Lavis (2006) Mr X ate Apple Breadand He suffered Xianethis Which illness followed Bread during training? Daryosis Xianethis Plonthema Eneuritis To what extent did the food cause this illness? 0 1 2 3 4 5 6 7 8 definitely

not causal possibly definitely

causal Blocking in ratings

of causality… … and in memory

of outcomes A → outcome AB → outcome B = Blocked CD → outcome D = Control Mitchell, Lovibond, Minard & Lavis (2006) Phase 1 Phase 2 Test A → outcome AB → outcome B? Blocking of memory Causality Memory Propositional account Blocking of causal

judgement Good memory of

outcome Link-based account

(e.g. RW) Blocking of causal

judgement Blocking of memory

of outcome Summary Propositions Links Backward

blocking Blocking of

memory of cue- outcome pairings Summary • It’s still not clear: More research needed! • Seems likely that both types of process operate • Simpler experiments encourage “thinking about it” • More complex experiments discourage thinking about it, so more

automatic link-formation mechanism dominates Wasserman & Berglan (1998) Backward blocking Supports propositional Mitchell et al (2006): Blocking of cue-outcome memory Supports link-based Summary • “Associative learning” may be due to more

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